Document Detail


Adult or late-onset triple A syndrome: case report and literature review.
MedLine Citation:
PMID:  20674935     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Triple A syndrome is caused by mutations in the gene encoding ALADIN, leading to achalasia, alacrima and addisonism. Neurologic manifestations of the disease include motor neuron disease-like presentations, motor-sensory or autonomic neuropathy, optic atrophy, cerebellar ataxia, Parkinsonism, and mild dementia. We report a 60-year-old Japanese man with triple A syndrome. He was born to non-consanguineous parents. He underwent a surgical operation for achalasia at age 40, and thereafter, he developed a slowly progressive gait disturbance. Neurological examinations at age 60 revealed limb muscle wasting and weakness with pyramidal tract signs, distal-dominant sensory disturbance, optic atrophy, and autonomic dysfunction. Alacrima was detected using Schirmer test. All of these features were consistent with typical triple A syndrome. He lacked adrenal insufficiency that is frequently observed in patients with the classic phenotype of triple A syndrome. His sural nerve biopsy showed a moderate loss of myelinated fibers and hypomyelination. He was homozygous for a missense mutation, p.R155H, in the disease-causing gene, AAAS. Seven patients with genetically-confirmed, adult or late-onset triple A syndrome, including ours, have been reported to date. All the patients showed upper and lower motor neuron signs (100%), while sensory disturbance (29%) and autonomic dysfunction (57%) were less frequent. Careful assessment for alacrima followed by molecular genetic analysis of AAAS should be considered in patients who show a combined phenotype of motor neuron disease and sensory/autonomic disturbance, even in elderly patients.
Authors:
Katsuya Nakamura; Kunihiro Yoshida; Tsuneaki Yoshinaga; Minori Kodaira; Yasuhiro Shimojima; Yo-Ichi Takei; Hiroshi Morita; Katsuhiko Kayanuma; Shu-Ichi Ikeda
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Publication Detail:
Type:  Case Reports; Journal Article; Review     Date:  2010-08-01
Journal Detail:
Title:  Journal of the neurological sciences     Volume:  297     ISSN:  1878-5883     ISO Abbreviation:  J. Neurol. Sci.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-09-13     Completed Date:  2010-12-28     Revised Date:  2011-12-06    
Medline Journal Info:
Nlm Unique ID:  0375403     Medline TA:  J Neurol Sci     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  85-8     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier B.V. All rights reserved.
Affiliation:
Department of Medicine (Neurology and Rheumatology), Shinshu University School of Medicine, 3-1-1 Asahi, Matsumoto 390-8621, Japan.
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MeSH Terms
Descriptor/Qualifier:
Adrenal Insufficiency* / genetics,  pathology,  physiopathology
DNA Mutational Analysis / methods
Esophageal Achalasia* / genetics,  pathology,  physiopathology
Humans
Male
Middle Aged
Muscle, Skeletal / pathology
Neural Conduction / physiology
Neurologic Examination / methods
Sural Nerve / physiopathology
Tongue / pathology

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