Document Detail


Acute effects of calvarial damage on dural mast cells, pial vascular permeability, and cerebral cortical histamine levels in rats and mice.
MedLine Citation:
PMID:  18355158     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Neurological complications after mild head injury can include vasogenic edema and/or subsequent development of epilepsy, conditions associated with elevated histamine. In the present study we assessed the potential of mast cells located in the dura mater to contribute to elevated cortical histamine and breakdown of the blood-brain barrier after minor head injury, modeled by either a parietal craniectomy or producing a groove in (scoring) the parietal bone surface to model a grazing head injury. We measured the following effects at 5-20 min after a unilateral parietal craniectomy (rats) or unilateral scoring of the parietal bone (mice): (1) mast cell integrity in subjacent dura mater; (2) subjacent vs. contralateral histamine in dura mater and cerebral cortex; (3) vascular permeability of cerebral cortical blood vessels subjacent to the injury, and; (4) the effects of an H(2)-receptor antagonist on cerebral cortical vascular permeability. RESULTS: Dural mast cells subjacent to the craniectomy became activated (degranulated) concomitant with (1) decreased histamine in dura mater subjacent to the craniectomy; (2) increased histamine in the subjacent cerebral cortex; and (3) extravasation of Evans blue-albumin which stained the subjacent cerebral cortex, indicating a localized breakdown of the blood-brain barrier. Similar results were observed in mice after scoring the parietal bone surface and, additionally, pretreatment with the histamine H(2)-receptor antagonist zolantadine (1 h before injury) dose-dependently inhibited extravasation of Evans blue-albumin. We conclude that even a minor grazing injury of the skull, in the absence of penetrating brain injury or concussion, can activate dural mast cells and elevate cortical histamine, a novel mechanism with potential contributions to neurotraumatic complications arising from a relatively minor or grazing head wound.
Authors:
Martha E Stokely; Edward L Orr
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of neurotrauma     Volume:  25     ISSN:  0897-7151     ISO Abbreviation:  J. Neurotrauma     Publication Date:  2008 Jan 
Date Detail:
Created Date:  2008-03-21     Completed Date:  2008-05-16     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8811626     Medline TA:  J Neurotrauma     Country:  United States    
Other Details:
Languages:  eng     Pagination:  52-61     Citation Subset:  IM    
Affiliation:
Department of Pharmacology and Neuroscience, and North Texas Eye Research Institute, University of North Texas Health Science Center at Fort Worth, Fort Worth, Texas, USA.
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MeSH Terms
Descriptor/Qualifier:
Albumins / diagnostic use,  pharmacokinetics
Animals
Blood-Brain Barrier / drug effects,  metabolism,  physiopathology*
Cerebral Arteries / metabolism,  physiopathology
Cerebral Cortex / blood supply,  metabolism*,  physiopathology
Chemotaxis / physiology
Craniocerebral Trauma / metabolism,  physiopathology
Craniotomy
Disease Models, Animal
Dose-Response Relationship, Drug
Dura Mater / cytology,  injuries*,  physiopathology*
Evans Blue / diagnostic use,  pharmacokinetics
Female
Head Injuries, Closed / metabolism,  physiopathology
Histamine / metabolism*
Histamine H2 Antagonists / pharmacology
Mast Cells / metabolism,  secretion*
Mice
Microcirculation / metabolism,  physiopathology
Pia Mater / blood supply
Rats
Rats, Inbred Lew
Secretory Vesicles / metabolism,  secretion
Skull / injuries*
Up-Regulation / physiology
Chemical
Reg. No./Substance:
0/Albumins; 0/Histamine H2 Antagonists; 314-13-6/Evans Blue; 51-45-6/Histamine

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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