| Acute congestive heart failure: pathophysiological alterations. | |
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MedLine Citation:
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PMID: 7670289 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The goal of a healthy heart is to transfer blood coming to the ventricles from the low pressure venous system into the high pressure arterial system. In acute congestive heart failure (CHF), impaired cardiac function leads to failure to empty venous reservoirs and therefore to reduce delivery of blood into the arterial circulation (Michaelson 1983). Once heart failure occurs it has a high mortality rate. According to the Framingham Heart Study, 50% of patients with New York Heart Association class II or III heart failure died within 5 years. With class IV patients, 50% survived less than 1 year (Cupples 1987). The purpose of this article is first to define acute CHF. Secondly, the pathophysiological alterations that occur as a result of strained and misdirected compensatory mechanisms will be discussed to help critical care nurses to recognise why certain patients are at risk of developing acute CHF. |
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Authors:
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R Dahlen; S L Roberts |
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Publication Detail:
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Type: Journal Article |
Journal Detail:
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Title: Intensive & critical care nursing : the official journal of the British Association of Critical Care Nurses Volume: 11 ISSN: 0964-3397 ISO Abbreviation: Intensive Crit Care Nurs Publication Date: 1995 Aug |
Date Detail:
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Created Date: 1995-10-19 Completed Date: 1995-10-19 Revised Date: 2007-11-15 |
Medline Journal Info:
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Nlm Unique ID: 9211274 Medline TA: Intensive Crit Care Nurs Country: SCOTLAND |
Other Details:
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Languages: eng Pagination: 210-6 Citation Subset: N |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Acute Disease Heart Failure / classification, mortality, physiopathology* Humans Severity of Illness Index Survival Rate |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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