Document Detail


Acid-base changes during complete brain ischemia.
MedLine Citation:
PMID:  2122556     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We examined the proposal that preischemic hyperglycemia causes exaggerated brain damage by decreasing intracellular or extracellular pH to below a specified threshold value. We also provide a critical appraisal of two related hypotheses. The first is that hyperglycemia enhances brain damage by causing excessive intraglial acidosis; the second, that the critical degree of acidosis is reached not during the ischemia but when recirculation is instituted. The following conclusions are drawn. First, the evidence is inconclusive in favor of marked compartmentation of H+ during ischemia, based on a discontinuous delta lactate/delta PCO2 relation and on direct intracellular pH measurements. In fact, results obtained with identical techniques in normoglycemic animals suggest that the acid compartment assumed to be glia is very small and may be of another origin. Second, although recirculation may give rise to a further increase in either extracellular or intracellular acidosis under certain conditions, this acidosis is not a prerequisite for increased tissue damage or infarction. Third, a critical appraisal of reports supports the contention that enhanced damage is triggered below a specified threshold pH value. In complete or near-complete ischemia, this value corresponds to a tissue lactate content of 17-20 mM.kg-1 wet wt. No correlation exists between subthreshold values for delta lactate and the severity of tissue damage. Furthermore, hyperglycemia cannot be expected to enhance damage if conditions prevent lactate from reaching threshold values or if they uncouple changes in lactate and pH.
Authors:
B K Siesjö; A Ekholm; K Katsura; S Theander
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Stroke; a journal of cerebral circulation     Volume:  21     ISSN:  0039-2499     ISO Abbreviation:  Stroke     Publication Date:  1990 Nov 
Date Detail:
Created Date:  1990-12-20     Completed Date:  1990-12-20     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0235266     Medline TA:  Stroke     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  III194-9     Citation Subset:  IM    
Affiliation:
Laboratory for Experimental Brain Research, University of Lund, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Acid-Base Equilibrium
Acidosis / metabolism*
Acidosis, Lactic / metabolism
Adenosine Triphosphate / metabolism
Bicarbonates / metabolism*
Brain Ischemia / metabolism*
Carbon Dioxide / metabolism
Electrolytes / metabolism
Extracellular Space / metabolism
Glucose / metabolism
Hydrogen-Ion Concentration
Hyperglycemia / metabolism
Neuroglia / metabolism
Neurons / metabolism
Grant Support
ID/Acronym/Agency:
5-RO1//PHS HHS
Chemical
Reg. No./Substance:
0/Bicarbonates; 0/Electrolytes; 124-38-9/Carbon Dioxide; 50-99-7/Glucose; 56-65-5/Adenosine Triphosphate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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