Document Detail


Accumulation of Ym1 and formation of intracellular crystalline bodies in alveolar macrophages lacking heparanase.
MedLine Citation:
PMID:  20226534     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Heparanase is a heparan sulfate (HS) degrading endoglucuronidase that has been implicated in cell migration and inflammatory conditions. Here we used mice deficient of heparanase (Hpse(-/-)) to study the impact of heparanase on airway leukocytes. Normal numbers of macrophages and lymphocytes were present in bronchoalveolar lavage fluid of Hpse(-/-) mice, indicating that heparanase is not essential for proper homing of leukocytes to airways. While lymphocytes from Hpse(-/-) mice displayed normal morphology, Hpse(-/-) alveolar macrophages showed a striking, age-dependent appearance of granule-like structures in the cytoplasm. Transmission electron microscopy revealed that these structures corresponded to membrane-enclosed crystalline bodies that closely resembled the intracellular crystals known to be formed by the HS-binding protein Ym1, suggesting that heparanase deficiency is associated with intracellular Ym1 deposition. Indeed, applying immunocytochemistry, we found markedly higher levels of intracellular Ym1 protein in Hpse(-/-) versus WT alveolar macrophages, and there was a significant correlation between levels of Ym1 protein detected by immunoblotting and amounts of crystalline material in BAL cells. Biosynthetic radio-labeling of the macrophages revealed accumulation of non-degraded HS chains in Hpse(-/-) macrophages. Together, these findings implicate heparanase in normal processing of HS in macrophages, and indicate that heparanase regulates intracellular Ym1 accumulation and crystal formation in airways.
Authors:
Ida Waern; Juan Jia; Gunnar Pejler; Eyal Zcharia; Israel Vlodavsky; Jin-Ping Li; Sara Wernersson
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-03-11
Journal Detail:
Title:  Molecular immunology     Volume:  47     ISSN:  1872-9142     ISO Abbreviation:  Mol. Immunol.     Publication Date:  2010 Apr 
Date Detail:
Created Date:  2010-04-05     Completed Date:  2010-04-22     Revised Date:  2011-12-07    
Medline Journal Info:
Nlm Unique ID:  7905289     Medline TA:  Mol Immunol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1467-75     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Department of Anatomy, Physiology and Biochemistry, Swedish University of Agricultural Sciences, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Line
Cell Shape
Crystallins / metabolism*
Glucuronidase / deficiency,  metabolism*
Intracellular Space / metabolism
Lectins / metabolism*
Macrophages, Alveolar / immunology,  metabolism*,  ultrastructure
Mice
Mice, Knockout
Microscopy, Electron, Transmission
beta-N-Acetylhexosaminidases / metabolism*
Grant Support
ID/Acronym/Agency:
R01 CA106456-09/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Crystallins; 0/Lectins; EC 3.2.1.-/heparanase; EC 3.2.1.31/Glucuronidase; EC 3.2.1.52/Chi3l3 protein, mouse; EC 3.2.1.52/beta-N-Acetylhexosaminidases

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