Document Detail


Abnormalities of aldosterone synthesis and action in children.
MedLine Citation:
PMID:  9300202     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Genetic defects in aldosterone biosynthesis and action affect blood pressure and electrolyte homeostasis. Aldosterone synthase deficiency, salt-wasting forms of congenital adrenal hyperplasia, and adrenal hypoplasia congenita all cause aldosterone deficiency, signs of which include hyponatremia, hyperkalemia, hypovolemia, elevated plasma renin activity, and sometimes shock and death. Conversely, the inappropriate regulation of aldosterone synthesis seen in glucocorticoid-suppressible hyperaldosteronism may cause hypokalemia, suppressed plasma renin activity, and hypertension. Similar problems occur when the normal ligand specificity of the aldosterone receptor is lost, as in the syndrome of apparent mineralocorticoid excess due to 11 beta-hydroxysteroid dehydrogenase deficiency. The effects of aldosterone are mediated largely through activation of the epithelial sodium channel, and inactivating or activating mutations of this channel leads to signs of mineralocorticoid deficiency or excess, termed pseudohypoaldosteronism and Liddle's syndrome, respectively.
Authors:
P C White
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Current opinion in pediatrics     Volume:  9     ISSN:  1040-8703     ISO Abbreviation:  Curr. Opin. Pediatr.     Publication Date:  1997 Aug 
Date Detail:
Created Date:  1997-10-14     Completed Date:  1997-10-14     Revised Date:  2005-11-16    
Medline Journal Info:
Nlm Unique ID:  9000850     Medline TA:  Curr Opin Pediatr     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  424-30     Citation Subset:  IM    
Affiliation:
UT Southwestern Medical Center, Dallas, TX 75235-9063, USA.
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MeSH Terms
Descriptor/Qualifier:
Adrenal Hyperplasia, Congenital / complications,  genetics*
Aldosterone Synthase / deficiency*
Child
Humans
Hyperaldosteronism / complications,  genetics*
Hypoaldosteronism / complications,  genetics*
Mutation / genetics
Pseudohypoaldosteronism / complications,  genetics*
Water-Electrolyte Imbalance / etiology
Chemical
Reg. No./Substance:
EC 1.14.15.4/Aldosterone Synthase
Comments/Corrections
Comment In:
Curr Opin Pediatr. 1997 Aug;9(4):407-12   [PMID:  9300199 ]

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