Document Detail


ASNA-1 activity modulates sensitivity to cisplatin.
MedLine Citation:
PMID:  20966125     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cancer can be cured by platinum-based chemotherapy, but resistance is a major cause of treatment failure. Here we present the nematode Caenorhabditis elegans as a model to study interactions between the platinum drug cisplatin and signaling pathways in vivo. Null mutation in a single gene, asna-1, makes worms hypersensitive to cisplatin. The metalloregulated ATPase ASNA-1 promotes insulin secretion and membrane insertion of tail-anchored proteins. Using structural data from ASNA-1 homologues, we identify specific ASNA-1 mutants that are sensitive to cisplatin while still able to promote insulin signaling. Mutational analysis reveals that hypersensitivity of ASNA-1 mutants to cisplatin remains in absence of CEP-1/p53 or apoptosis. Human ASNA1 can substitute for the worm gene, indicating a conserved function. Cisplatin sensitivity is not affected by decreased insulin signaling in wild-type nematodes or restored insulin signaling in asna-1 mutants. These findings provide a functional insight into ASNA-1, demonstrate that C. elegans can be used to characterize cisplatin resistance mechanisms, and suggest that rationally designed drugs against ASNA-1 can sensitize cancer cells to cisplatin.
Authors:
Oskar Hemmingsson; Gautam Kao; Maria Still; Peter Naredi
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-10-21
Journal Detail:
Title:  Cancer research     Volume:  70     ISSN:  1538-7445     ISO Abbreviation:  Cancer Res.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-12-16     Completed Date:  2011-01-20     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  2984705R     Medline TA:  Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  10321-8     Citation Subset:  IM    
Copyright Information:
©2010 AACR.
Affiliation:
Division of Surgery, Department of Surgical and Perioperative Sciences, Umeå University, Umeå, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects,  genetics
Arsenite Transporting ATPases / genetics*
Caenorhabditis elegans / genetics,  metabolism
Caenorhabditis elegans Proteins / genetics*
Cell Growth Processes / drug effects,  genetics
Cisplatin / pharmacology*
Humans
Insulin / metabolism
Mutagenesis, Site-Directed
Signal Transduction / drug effects
Chemical
Reg. No./Substance:
0/ASNA1 protein, human; 0/Caenorhabditis elegans Proteins; 11061-68-0/Insulin; 15663-27-1/Cisplatin; EC 3.6.3.16/ASNA-1 protein, C elegans; EC 3.6.3.16/Arsenite Transporting ATPases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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