Document Detail


AMPA receptor trafficking in inflammation-induced dorsal horn central sensitization.
MedLine Citation:
PMID:  22466122     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Activity-dependent postsynaptic receptor trafficking is critical for long-term synaptic plasticity in the brain, but it is unclear whether this mechanism actually mediates the spinal cord dorsal horn central sensitization (a specific form of synaptic plasticity) that is associated with persistent pain. Recent studies have shown that peripheral inflammation drives changes in α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPAR) subunit trafficking in the dorsal horn and that such changes contribute to the hypersensitivity that underlies persistent pain. Here, we review current evidence to illustrate how spinal cord AMPARs participate in the dorsal horn central sensitization associated with persistent pain. Understanding these mechanisms may allow the development of novel therapeutic strategies for treating persistent pain.
Authors:
Yuan-Xiang Tao
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Neuroscience bulletin     Volume:  28     ISSN:  1995-8218     ISO Abbreviation:  Neurosci Bull     Publication Date:  2012 Apr 
Date Detail:
Created Date:  2012-04-02     Completed Date:  2012-12-06     Revised Date:  2013-04-03    
Medline Journal Info:
Nlm Unique ID:  101256850     Medline TA:  Neurosci Bull     Country:  China    
Other Details:
Languages:  eng     Pagination:  111-20     Citation Subset:  IM    
Affiliation:
Department of Anesthesiology and Critical Care Medicine, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205, USA. ytao1@jhmi.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Chronic Pain / etiology,  physiopathology*
Glutamic Acid / physiology
Humans
Inflammation / complications,  physiopathology*
Neuronal Plasticity / physiology
Nociceptors / physiology*
Posterior Horn Cells / physiopathology*
Protein Transport / physiology
Receptors, AMPA / physiology*
Synaptic Transmission / physiology
Grant Support
ID/Acronym/Agency:
NS058886/NS/NINDS NIH HHS; NS072206/NS/NINDS NIH HHS; R01 NS058886-04/NS/NINDS NIH HHS; R01 NS072206-01A1/NS/NINDS NIH HHS; R01 NS072206-02/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Receptors, AMPA; 56-86-0/Glutamic Acid

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