Document Detail


AID produces DNA double-strand breaks in non-Ig genes and mature B cell lymphomas with reciprocal chromosome translocations.
MedLine Citation:
PMID:  19941823     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Cancer-initiating translocations such as those associated with lymphomas require the formation of paired DNA double-strand breaks (DSBs). Activation-induced cytidine deaminase (AID) produces widespread somatic mutation in mature B cells; however, the extent of "off-target" DSB formation and its role in translocation-associated malignancy is unknown. Here, we show that deregulated expression of AID causes widespread genome instability, which alone is insufficient to induce B cell lymphoma; transformation requires concomitant loss of the tumor suppressor p53. Mature B cell lymphomas arising as a result of deregulated AID expression are phenotypically diverse and harbor clonal reciprocal translocations involving a group of Immunoglobulin (Ig) and non-Ig genes that are direct targets of AID. This group includes miR-142, a previously unknown micro-RNA target that is translocated in human B cell malignancy. We conclude that AID produces DSBs throughout the genome, which can lead to lymphoma-associated chromosome translocations in mature B cells.
Authors:
Davide F Robbiani; Samuel Bunting; Niklas Feldhahn; Anne Bothmer; Jordi Camps; Stephanie Deroubaix; Kevin M McBride; Isaac A Klein; Gary Stone; Thomas R Eisenreich; Thomas Ried; André Nussenzweig; Michel C Nussenzweig
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Molecular cell     Volume:  36     ISSN:  1097-4164     ISO Abbreviation:  Mol. Cell     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-11-27     Completed Date:  2009-12-16     Revised Date:  2011-05-30    
Medline Journal Info:
Nlm Unique ID:  9802571     Medline TA:  Mol Cell     Country:  United States    
Other Details:
Languages:  eng     Pagination:  631-41     Citation Subset:  IM    
Affiliation:
The Rockefeller University, New York, NY 10065, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
B-Lymphocytes / cytology,  enzymology
Cell Differentiation / genetics
Cells, Cultured
Chromosomal Instability / genetics
Chromosomes, Mammalian / genetics*
Cytidine Deaminase / metabolism*
DNA Breaks, Double-Stranded*
DNA Damage
Genes, Immunoglobulin / genetics*
Humans
Immunoglobulin Class Switching / genetics
Karyotyping
Lymphoma, B-Cell / enzymology*,  genetics*,  pathology
Mice
Mice, Transgenic
MicroRNAs / metabolism
Phenotype
Proto-Oncogene Proteins c-myc / genetics
Somatic Hypermutation, Immunoglobulin / genetics
Translocation, Genetic*
Tumor Suppressor Protein p53 / deficiency
Grant Support
ID/Acronym/Agency:
AI037526/AI/NIAID NIH HHS; GM07739/GM/NIGMS NIH HHS; R01 AI037526-16/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/MicroRNAs; 0/Mirn142 microRNA, mouse; 0/Proto-Oncogene Proteins c-myc; 0/Tumor Suppressor Protein p53; EC 3.5.4.-/AICDA (activation-induced cytidine deaminase); EC 3.5.4.5/Cytidine Deaminase
Comments/Corrections

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