Document Detail


AICA-riboside induces apoptosis of pancreatic beta cells through stimulation of AMP-activated protein kinase.
MedLine Citation:
PMID:  12627324     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
AIMS/HYPOTHESIS: Prolonged exposure of beta cells to low glucose concentrations triggers their apoptosis and is known to activate AMP-activated protein kinase (AMPK) in beta cell lines. We examined whether prolonged activation of AMPK can trigger apoptosis in rodent beta cells. METHODS: Primary beta cells were FACS-purified from rats, and from wild-type and AMPK(alpha2)-deficient mice. AMPK activation in beta cells was induced by the adenosine analog AICA-riboside and detected by immunoblotting using a phosphospecific antibody. Apoptosis of rodent beta cells was monitored by FACS analysis of beta cell DNA content, by direct counting of apoptotic cells using fluorescence microscopy, or by measurement of their caspase-3 activity. RESULTS: Dose-dependent and time-dependent apoptosis of the cells, concommittant with an activation of caspase-3, were suppressed by the caspase inhibitors zVAD-fmk and zDEVD-fmk. Apoptosis induction by AICA-riboside was also prevented by adding the MAPK-inhibitor SB203580 which blocked the AICA-riboside-induced phosphorylation of AMPK. Beta cells isolated from AMPK-(alpha2)-deficient mice were resistant against AICA-riboside induced apoptosis. CONCLUSION/INTERPRETATION: Sustained activation of AMPK by AICA-riboside can trigger a caspase-dependent apoptosis of pancreatic beta cells.
Authors:
B A Kefas; H Heimberg; S Vaulont; D Meisse; L Hue; D Pipeleers; M Van de Casteele
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2003-02-08
Journal Detail:
Title:  Diabetologia     Volume:  46     ISSN:  0012-186X     ISO Abbreviation:  Diabetologia     Publication Date:  2003 Feb 
Date Detail:
Created Date:  2003-03-10     Completed Date:  2003-12-05     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  0006777     Medline TA:  Diabetologia     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  250-4     Citation Subset:  IM    
Affiliation:
Diabetes Research Center, Free Brussels University-VUB, Laarbeeklaan 103, 1090 Brussels, Belgium.
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MeSH Terms
Descriptor/Qualifier:
AMP-Activated Protein Kinases
Aminoimidazole Carboxamide / analogs & derivatives*,  pharmacology*
Animals
Apoptosis*
Caspase 3
Caspases / metabolism
Cells, Cultured
Enzyme Activation
Islets of Langerhans / drug effects*,  physiology*
Mice
Multienzyme Complexes / deficiency,  metabolism*
Protein-Serine-Threonine Kinases / deficiency,  metabolism*
Rats
Ribonucleosides / pharmacology*
Chemical
Reg. No./Substance:
0/Multienzyme Complexes; 0/Ribonucleosides; 2627-69-2/acadesine; 360-97-4/Aminoimidazole Carboxamide; EC 2.7.11.1/AMP-Activated Protein Kinases; EC 2.7.11.1/Protein-Serine-Threonine Kinases; EC 3.4.22.-/Casp3 protein, mouse; EC 3.4.22.-/Casp3 protein, rat; EC 3.4.22.-/Caspase 3; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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