Document Detail


ACTH-(1-24) blocks the decompensatory phase of the haemodynamic response to acute hypovolaemia in conscious rabbits.
MedLine Citation:
PMID:  7768294     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Graded caval occlusion in conscious rabbits caused a biphasic cardiovascular response. Phase I was characterized by a fall in systemic vascular conductance so that arterial pressure was maintained. When cardiac output had fallen to 64 +/- 3% of its baseline level, phase II supervened. During phase II, conductance rose abruptly and arterial pressure fell to a life-threatening level (< 40 mm Hg). Intravenous (i.v.) or central (fourth ventricular) administration of the adrenocorticotrophin (ACTH) fragment ACTH-(1-24) prevented the occurrence of phase II. The central dose of ACTH-(1-24) needed to block the occurrence of phase II was approximately 39 times less than the i.v. dose. Central administration of the delta 1-opioid receptor agonist [D-Pen2,D-Pen5]enkephalin (DPDPE) reversed this effect of both central and i.v. ACTH-(1-24). I.v. ACTH-(1-24) also lowered arterial pressure while raising cardiac output and vascular conductance. These effects were insensitive to propranolol and hyoscine methyl bromide, and were not mimicked by cortisol or adrenaline. It is concluded that ACTH-(1-24) has an acute, adrenal-independent, peripheral vasodilator effect as well as a central, anti-shock, effect.
Authors:
J Ludbrook; S Ventura
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  European journal of pharmacology     Volume:  275     ISSN:  0014-2999     ISO Abbreviation:  Eur. J. Pharmacol.     Publication Date:  1995 Mar 
Date Detail:
Created Date:  1995-06-30     Completed Date:  1995-06-30     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  1254354     Medline TA:  Eur J Pharmacol     Country:  NETHERLANDS    
Other Details:
Languages:  eng     Pagination:  267-75     Citation Subset:  IM    
Affiliation:
University of Melbourne, Department of Surgery, Royal Melbourne Hospital, Parkville, Victoria, Australia.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cosyntropin / pharmacology*
Enkephalin, D-Penicillamine (2,5)-
Enkephalins / pharmacology
Epinephrine / pharmacology
Hemodynamics / drug effects*
Hydrocortisone / pharmacology
Nitroprusside / pharmacology
Propranolol / pharmacology
Rabbits
Scopolamine / pharmacology
Shock / physiopathology*
Chemical
Reg. No./Substance:
0/Enkephalins; 15078-28-1/Nitroprusside; 16960-16-0/Cosyntropin; 50-23-7/Hydrocortisone; 51-34-3/Scopolamine; 51-43-4/Epinephrine; 525-66-6/Propranolol; 88373-73-3/Enkephalin, D-Penicillamine (2,5)-

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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