Document Detail


2-(4-Chlorophenyl)benzo-1,4-quinone induced ROS-signaling inhibits proliferation in human non-malignant prostate epithelial cells.
MedLine Citation:
PMID:  20163859     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Polychlorinated biphenyls (PCBs) and their metabolites are environmental chemical contaminants which can produce reactive oxygen species (ROS) by auto-oxidation of di-hydroxy PCBs as well as the reduction of quinones and redox-cycling. We investigate the hypothesis that 2-(4-chlorophenyl)benzo-1,4-quinone (4-Cl-BQ), a metabolite of 4-chlorobiphenyl (PCB3), induced ROS-signaling inhibits cellular proliferation. Monolayer cultures of exponentially growing asynchronous human non-malignant prostate epithelial cells (RWPE-1) were incubated with 0-6 μM of 4-Cl-BQ and harvested at the end of 72 h of incubation to assess antioxidant enzyme expression, cellular ROS levels, cell growth, and cell cycle phase distributions. 4-Cl-BQ decreased manganese superoxide dismutase (MnSOD) activity, protein, and mRNA levels. 4-Cl-BQ treatment increased dihydroethidium (DHE) fluorescence, which was suppressed in cells pretreated with polyethylene glycol conjugated superoxide dismutase (PEG-SOD). The increase in ROS levels was associated with a decrease in cell growth, and an increase in the percentage of S-phase cells. These effects were suppressed in cells pretreated with PEG-SOD. 4-Cl-BQ treatment did not change the protein levels of phosphorylated H2AX at the end of 72 h of incubation, suggesting that the inhibition in cell growth and accumulation of cells in S-phase at the end of the treatments were probably not due to 4-Cl-BQ induced DNA double strand break. These results demonstrate that MnSOD activity and ROS-signaling perturb proliferation in 4-Cl-BQ treated in vitro cultures of human prostate cells.
Authors:
Leena Chaudhuri; Ehab H Sarsour; Prabhat C Goswami
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-02-16
Journal Detail:
Title:  Environment international     Volume:  36     ISSN:  1873-6750     ISO Abbreviation:  Environ Int     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-10-04     Completed Date:  2011-01-11     Revised Date:  2011-11-01    
Medline Journal Info:
Nlm Unique ID:  7807270     Medline TA:  Environ Int     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  924-30     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ltd. All rights reserved.
Affiliation:
Free Radical and Radiation Biology Program, Department of Radiation Oncology, University of Iowa, Iowa City, Iowa, United States.
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MeSH Terms
Descriptor/Qualifier:
Biphenyl Compounds / toxicity*
Cell Proliferation / drug effects*
Cells, Cultured
Epithelial Cells / drug effects*
Humans
Male
Prostate / drug effects
Quinones / toxicity*
Reactive Oxygen Species / metabolism*
Signal Transduction*
Superoxide Dismutase / metabolism
Grant Support
ID/Acronym/Agency:
CA 111365/CA/NCI NIH HHS; P42 ES 013661/ES/NIEHS NIH HHS; P42 ES013661-040002/ES/NIEHS NIH HHS; R01 CA111365-05/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Biphenyl Compounds; 0/Quinones; 0/Reactive Oxygen Species; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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