Conservative treatment of a tibialis posterior strain in a novice triathlete: a case report.
Objective: To detail the progress of a novice triathlete with an
unusual mechanism of a tibialis posterior strain who underwent
successful conservative treatment and rehabilitation. Tibialis posterior
tendon dysfunction will be discussed as it relates to the case.
Clinical Features: The clinical features of tibialis posterior dysfunction are swelling and edema posterior to the medial malleolus with pain and an inability to weight bear. This injury may occur in endurance athletes such as triathletes, most often occurring during running.
Intervention and Outcome: The conservative treatment approach used in this case consisted of medical acupuncture with electrical stimulation, Graston Technique[c] a soft tissue instrument assisted mobilization technique, Active Release Technique[R], ultrasound therapy with Traumeel, and rehabilitation. Gait analysis and orthotic prescription was completed when the patient was ready to return to play. Outcome measures included subjective pain rating and return to pre-injury activities. Objective measures included swelling and manual muscle testing.
Conclusion: A novice triathlete with a grade I tibialis posterior strain was quickly relieved of his symptoms and able to return to his triathlon training with conservative treatment. Practitioners treating this type of injury could consider including the soft tissue techniques, modalities and rehabilitation employed in our case for other patients with lower leg strains and/or tibialis posterior dysfunction. (JCCA 2009; 53(1):23-31)
KEY WORDS: tibialis posterior strain, Active Release Technique[R], Graston Technique[c], triathlete
Objectif: Enumerer lesprogres d'un jeune triathlonien affichant un mecanisme rare d'une elongation du jambier posterieur traite avec succes par une methode conventionnelle, puis une readaptation. Nous commenterons la problematique du dysfonctionnement du tendon du jambier posterieur, qui se rapporte au present cas.
Caracteristiques cliniques: Les caracteristiques cliniques du dysfonctionnement du jambier posterieur se manifestent par une tumefaction et un cedeme posterieur de la malleole interne, marques par la douleur et une incapacite de supporter un poids. Cette blessure se presente chez les athletes qui pratiquent des sports d'endurance, par exemple le triathlon, et survient la plupart du temps pendant la course.
Intervention et resultat: L'approche conventionnelle du traitement dans le present cas a consiste a avoir recours a l'acupuncture medicale avec une stimulation electrique, la technique Graston[R], une technique de mobilisation assistee par un instrument pour tissus mous, l'Active Release Technique[R] (technique de relachement active), l'ultrasonotherapie avec le medicament regulateur de l'inflammation Traumeel et la readaptation. Une fois le malade pret a retourner au jeu, on a procede a une analyse de la demarche et prescrit une orthese. Dans les indicateurs de resultats, on compte un niveau subjectif de la douleur et le retour aux activites pratiquees avant la blessure. Les mesures objectives comprenaient l'inflammation et un test fonctionnel du bilan musculaire.
Conclusion: Un jeune triathlonien affecte d'une elongation du jambier posterieur de premier stade a ete rapidement soulage de ses symptomes et en mesure de retourner a l'entratnement grace a un traitement conventionnel. Les praticiens ayant a soigner ce type de blessures peuvent envisager d'inclure des techniques de traitement des tissus mous, des methodes et une readaptation utilisees dans le present cas pour d'autres malades souffrant de blessures dans la partie inferieure de la jambe et/ou d'un dysfonctionnement du jambier posterieur. (JACC 2009; 53(1):23-31)
MOTS CLES: elongation du jambier posterieur, Active Release Technique[R], technique Graston[R], triathlete
|Article Type:||Case study|
Sprains (Care and treatment)
Sprains (Case studies)
Sprains (Risk factors)
Rehabilitation (Health aspects)
|Publication:||Name: Journal of the Canadian Chiropractic Association Publisher: Canadian Chiropractic Association Audience: Academic Format: Magazine/Journal Subject: Health Copyright: COPYRIGHT 2009 Canadian Chiropractic Association ISSN: 0008-3194|
|Issue:||Date: Jan, 2009 Source Volume: 53 Source Issue: 1|
|Geographic:||Geographic Scope: Canada Geographic Code: 1CANA Canada|
Lower limb strain injuries are a common complaint that present to chiropractic and sports injury clinics. This area of complaint is especially common among triathletes because of the repetitive motions that occur while training for increased endurance in each sport (swimming, cycling and running). The most common injuries that occur in triathletes are a result of overuse. (1,2,3,4) The repetitive motions that are indicative of endurance training invariably put the triathlete at an increased risk of suffering an overuse injury. Most triathletes follow a periodized training schedule to achieve a balance between intense workouts and base or tempo workouts to avoid the effects of overtraining. The majority of triathletes have a previous running background, however, overtraining associated with high volumes of running mileage is reportedly common. (2,3)
Epidemiology studies note that the majority of triathlon-related injuries occur during run training and affect the lower limb. (3) This is thought to be a result of poor running mechanics and/or training errors which may involve increasing mileage too rapidly, speed training, and hill training. (3) These lower limb injuries are further confounded by cycling training which requires a continual effort from the gastrocsoleus group to generate power during pedaling. This cumulative effect may lead the athlete to be more susceptible to overuse trauma during running. (1,2)
The majority of literature on triathlon injuries has focused on the most common injuries that occur during running. The incidence of swimming-related injuries amongst triathletes is relatively low, despite the relative inexperience in swimming for the majority of triathletes. (3)
Injuries directly related to swimming typically involve the shoulder and are primarily tendon or impingement-related diagnoses. (3) Although the shoulder is the most common area injured during swimming, injuries indirectly related to swimming include lower leg injuries as posterior calf tightness is associated with pointing the toes during leg kicking for swim propulsion. (3) While swimming, the feet are maintained in a plantar flexed position for a more streamlined position and this plantar flexed position promotes a relative shortening of the posterior calf musculature.
A 41-year old male novice triathlete training for an Ironman distance triathlon, presented with a chief complaint of acute right ankle pain. The onset was three days prior while performing a flip-turn in a swimming pool during a training swim. Some discomfort developed in the right calf about an hour into the swim and just before the end of the session he felt extreme right calf pain when he pushed off the edge of the pool; there was no pop or snap reported but he had an immediate cramping sensation. He stopped swimming at that point and was able to walk for the rest of the day with only mild discomfort. The following day the calf worsened as there was significant swelling posterior to the right medial malleolus which he attempted to control with a tensor wrap. The patient rated the initial intensity of the pain as eight out of ten (on a ten point scale) and described it as an achy sensation. Aggravating activities included climbing stairs and driving his car, both of which he avoided due to pain.
[FIGURE 1 OMITTED]
The ankle pain progressively worsened over the first three days after onset, to the point that upon presentation to a multidisciplinary sports injury clinic he had a significant limp as he avoided putting any pressure on the right foot and kept his right foot dangling in a plantar flexed position. This patient had been treated three months prior for a distal iliotibial band (ITB) friction syndrome and a gluteus medius weakness/dysfunction. Treatment for this former complaint included Active Release Techniques[R] (ART[R]), Graston Technique[c], acupuncture and gluteal strengthening exercises. He continued to train while managing the ITB injury focusing on his swimming technique, and cycling endurance, with a decreased frequency and duration of his running to avoid further exacerbation. His lateral knee complaint was improving with each successive treatment and was nearly 100% resolved when he suffered the new complaint.
On examination he was unable to bear weight through his right leg and refused the challenge to attempt a "toeoff" or toe walk. Palpation of the tibialis posterior tendon reproduced his extreme discomfort as did resisted plantar flexion and inversion. Passive dorsiflexion and eversion also caused a great deal of pain (all rated as eight out of ten). Gastrocnemius and soleus palpation was unremarkable and although palpation of the medial Achilles tendon was somewhat discomforting it did not reproduce his chief complaint. Ankle ligamentous stress testing was unremarkable. Swelling and discoloration were significant [Figure 1]. Girth measurement was 29 cm around the right lower leg, compared to 25.5 cm around the left.
[FIGURE 2 OMITTED]
The working diagnosis was a 1st degree tibialis posterior strain. Initial treatment consisted of medical acupuncture (4 points surrounding the injury) with electrical stimulation (IC-1107+ at 2 Hz frequency), therapeutic ultrasound with Traumeel (5 minutes, 50% duty cycle, 1 MHz frequency, 0.7 W intensity), Active Release Technique[R] (ART[R]) of gastrocnemius, soleus, and tibialis posterior muscles above and below the injury, and Graston Technique[c] soft tissue mobilization with GT 6 and GT 2 [Figure 2] posterior to the medial malleolus followed by ten minutes of ice and elevation. Five days after the injury, the athlete was assessed by a sports specialist medical physician who agreed with the working diagnosis of a calf strain prescribing Celebrex (200 mg) for the inflammation and ordering a diagnostic ultrasound to grade the injury.
Eight days after the initial presentation at the third treatment, the discoloration had noticeably improved and the right lower leg girth had improved to 27 cm. At this point the patient was bearing weight and walking with a deliberate gait without a compensatory limp. The pain progressed from being a constant ache to an occasional jab (rated as six out of ten) when he "stepped down the wrong way" (excessive pronation/eversion noted in stair decent). By the fourth visit (thirteen days later) the pain was rated "two or three" out of ten and the inflammation continued to improve. He no longer walked with any limp, could climb and descend stairs pain-free, and demonstrated that he only felt the pain when "planting his foot and rotating" (internal tibial rotation). Examination revealed residual mild tenderness with palpation of the tibialis posterior tendon and myotendinous junction with resisted plantar flexion and inversion testing causing minimal pain. At this point, he had returned to training in the pool, using a pullbuoy to minimize kicking and avoided pushing off the wall to change direction. He also had resumed his cycling training and reported no aggravation of ankle symptoms.
[FIGURE 3 OMITTED]
The patient could not attend treatment for the next two weeks due to work travel, but returned one month postinjury with the results of the diagnostic ultrasound that showed limited inflammation and no further evidence of muscular tear, ruling out any damage beyond the 1st degree strain. Further examination found no evidence of inflammation and only "slight" tenderness at the tibialis posterior tendon just posterior to the medial malleolus. At this point, he was instructed to continue his pre-injury training schedule: biking (three times per week), swimming (three times per week) with the modification of a pullbuoy, and was given tibialis posterior strengthening exercises of "tib post heel-ups" with a tennis ball squeezed between the medial malleoli [Figure 3]. One week later he returned with virtually no tenderness (less than one out of ten), full range of motion, full strength, and an otherwise resolved tibialis posterior strain.
Six weeks after the initial presentation the patient returned feeling 100% and reported no pain in or around the medial malleolus and no recurrences. He had continued with his biking and swimming program which had significantly progressed in distance and intensity, and he showed an eagerness to return to running. A gait analysis was performed to assess his running mechanics and revealed his natural "preferred" running cadence to be 80 strides per minute at 6 mph. He ran with excessive anterior lean of his upper body and a decreased amount of hip flexion/extension and knee flexion. His right foot showed increased pronation with a toeing-out / eversion positioning and he was noted to catch his right heel occasionally on his left calf when swinging his right leg anterior. Further analysis of his lower limb function showed a joint coupling dysfunction with single leg squat (excessive internal tibial rotation), excessive pronation while weightbearing with a low arch height, and calluses were noted under his metatarsal heads on both feet with dropped metatarsal heads evident. Ankle dorsiflexion was adequate, general foot motion was normal, and proprioception was unremarkable. A gait scan confirmed clinical findings of over-pronation and showed excessive weight (pressure) being absorbed through his right foot. Orthotics were prescribed and the patient was advised to alter his technique by increasing his knee and hip flexion through running drills, and to increase his cadence to 90+. The increased hip and knee motion and cadence significantly improved his foot position during foot strike and swing.
At follow-up, one month later, the patient reported no further tibialis posterior discomfort. He was running approximately three hours per week easily, at an improved cadence of 90, with a comfortable/efficient body position. The patient had also consulted his coach and was following our advice to gradually build up the duration and intensity of his runs to avoid any further aggravation or incidental injury.
[FIGURE 4 OMITTED]
Due to the physical demands of triathlon, it is not surprising that triathletes sustain a high number of overuse injuries. Burns et al. found that during a six-month training period, 50.4% of triathletes were injured. (1) This is consistent with other studies that found that 75% of injuries experienced by triathletes occurred during training and 78.9% were defined as overuse. (2) The most commonly reported injuries amongst triathletes are the ankle/foot, thigh, knee, lower leg, and the back. The frequency of lower extremity injuries is not surprising when considering the repetitive impact of weight-bearing forces associated with running and the extensive use of the lower extremities in cycling. (3) Overall, the majority of injuries occurred during running training while swimming and cycling were associated with a lower numbers of injuries. (1,3,4) The patient in this case report sustained an acute lower leg/ankle injury during a swimming training session which represents an uncommon mechanism of a lower limb injury for a triathlete. As previously mentioned, the incidence of swimming-related injuries is low and most of these injuries involve the shoulder. (3) Injuries related to swimming that do not commonly occur in the pool may include achilles tendonopathy, gastroc/soleus muscle strains, and tibialis posterior dysfunction due the position of the ankle and foot during the swim. This shortening or tightness in the calf can increase the susceptibility of the triathlete to overstress of these lower leg tendons. (3)
[FIGURE 5 OMITTED]
The tibialis posterior muscle originates on the posterior aspect of the tibia, fibula, and the interosseous membrane. It courses posteriorly and medially around the ankle in a groove adjacent to the medial malleolus and inserts on the midfoot in the area of the navicular tuberosity. (5,6) The medial malleolus serves to change the direction of pull on the tendon. [Figure 4 and 5] This is believed to increase the stresses on the tendon as rupture usually occurs in this area. (5,7) The tibialis posterior functions as a plantar flexor of the ankle and an inverter of the subtalar joint complex.
The tibialis posterior muscle initiates the process of inversion of the hindfoot during gait bringing it into a neutral position. This muscle truly drives the position of the hindfoot and determines the flexibility of the foot by its control over the transverse tarsal joints. The loss of the inversion force of the muscle explains why patients with tibialis posterior tendon injuries have only a limited ability, or are completely unable, to rise onto their toes from a position of single-leg stance. (6) While an acute tibialis posterior strain is uncommonly reported, the mechanism of the strain is relatively straightforward as a force imparted into the muscle exceeds its strength. On the other hand, the etiology of the more commonly investigated and reported tibialis posterior tendon dysfunction remains somewhat unclear and may include vascular, metabolic, or mechanical factors. (8) Dysfunction of the tibialis posterior tendon is a common cause of acquired adult flatfoot deformity (AFFD). (5) Middle age women are most commonly affected and the incidence is known to increase with age. Pes planus, hypertension, diabetes mellitus, and seronegative arthropathies have all been identified as risk factors for tibialis posterior dysfunction. (9)
Patients with tibialis posterior injuries will typically present with an insidious onset of vague pain in the medial foot and swelling behind the medial malleolus along the course of the tendon. Roughly half of all patients have a history of trauma that was initially thought to be a sprain. (5,6) Symptoms are usually aggravated by standing and walking and in addition to pain patients often note dysfunction in their gait. Typically these patients are unable to run and note difficulty taking a long stride as well as they have an inability to push off onto their toes and raise their heel. Some authors describe patients with tibialis posterior dysfunction presenting simply with pain and apparent inflammation along the tendon without any evidence of clinical deformity but most patients have some collapse of the foot. (6,9) Kohls-Gatzoulis et al. found that the complaint of medial pain or swelling behind the medial malleolus together with a change in foot shape identified 100% of patients with tibialis posterior dysfunction and had a specificity of 98%. (9)
Typically the physical examination of tibialis posterior dysfunction patients reveals a flatfoot deformity that consists of flattening of the medial longitudinal arch, hindfoot valgus, and abduction of the midfoot on the hindfoot. This abduction allows relatively more toes to be seen when standing behind the patient leading to the "too many toes" sign which is characterized by this condition. (5,6,9,10) Patients typically have a flatfooted heel-toe progression and a poor or absent heel rise, in fact those with a dysfunctional tibialis posterior muscle asked to rise onto their toes from a position of single-leg stance are either completely unable to comply or can do so only to a limited degree. (5,6,9) Ranges of motion are typically full in earlier stages of the condition and as the condition progresses the joints can lose motion and may eventually become fixed. (5,9)
Manual muscle testing of the tibialis posterior is performed by placing the foot in an everted, plantar flexed position and the patient is asked to invert the foot. Weakness or pain during contraction of an injured tibialis posterior muscle is characteristic. Palpation usually reveals tenderness along the distal aspect of the posterior tibial tendon from the medial malleolus to the navicular tuberosity; however, tenderness to palpation proximally along the musculotendinous junction of the tibialis posterior muscle may also be present in muscle strains. An accurate diagnosis of tibialis posterior tendon dysfunction can usually be made through a simple clinical examination however radiographic evaluation may be helpful in determining the severity of the condition or osseous involvement. Radiographic evaluation should include four weight-bearing films: an anteroposterior view of both ankles, an anteroposterior view of both feet, and lateral foot and ankle views of each side to allow comparison in patients who have unilateral disease. Typical deformity includes apparent shortening of the hindfoot on the weight-bearing anteroposterior radiograph, which is indicative of collapse through the subtalar joint complex. On the weight-bearing lateral radiographs, the inclination of the talus is plantarward in comparison to normal, with collapse typically through the talonavicular joint. (6) A more overt muscle strain or tendonopathy would easily be visualized through a diagnostic ultrasound which would also be useful for grading the injury or quantifying any underlying inflammation.
Johnson and Strom (11) initially described a classification scheme for tibialis posterior tendon insufficiency which was added to by Myerson. (12) [See Table 1] Although the classification is not predictive and does not consider the contracted gastrocnemius, the three stage scheme is useful for considering treatment strategies. In many ways, an acute tibialis posterior strain would be typical of stage 1 or stage 2 in this classification system.
It has been noted that there are difficulties with this classification systems reliability, making it somewhat difficult to compare results from various studies that use this system.
The tibialis posterior acts as a heel inverter creating an obliquity of the transverse tarsal joint, thereby allowing for a rigid midfoot during terminal stance, which in turn allows efficient transfer of stored energy in the lower extremity for toe-off and swing phase. (6,13) Therefore, dysfunction of the tibialis posterior muscle results in less efficient gait, as the heel does not effectively medialize, and the gastrocsoleus complex requires greater excursion to become a heel inverter. (13) Theoretically, rearfoot eversion and an increased medial longitudinal arch angle move the talonavicular and calcaneocuboid joints to a more parallel position, unlocking the foot for shock absorption. Gait patterns in normal subjects progress from a neutral (or slightly inverted position) to eversion at foot flat, the role of shock absorption is linked to these foot kinematics. While normal subjects increase rearfoot eversion and medial longitudinal arch angle throughout the stance, the subjects with posterior tibial tendon dysfunction are at, or near, peak rearfoot eversion and medial longitudinal arch angle during loading response. This failure of gradual shock absorption to occur may contribute to abnormal stresses on secondary ligamentous support (spring ligament, plantar fascia, interosseous talocalcaneal ligament) as the foot is loaded. During the terminal stance and preswing phases of gait, abnormal kinematics of the patients with posterior tibial tendon dysfunction suggests a failure to position the foot effectively for push off. (14)
Non-operative management of tibialis posterior injuries focuses on improving a patient's symptoms, usually by attempting to decrease the forces which pass through the posteromedial hindfoot. Any acute inflammation surrounding the sheath of the tibialis posterior tendon should be dealt with before the chronic aspect of the condition is treated.9 Non-steroidal anti-inflammatory medication may decrease pain and associated swelling, however, the initial conservative treatment of acute injuries of the tibialis posterior dysfunction is not unlike any other muscle strain and should include P.R.I.C.E. principles: Protection, Relative Rest (allowing as much motion and activity as possible to counter the deleterious effects of disuse while not unnecessarily stressing the healing tissues), Ice, Compression, and Elevation. Conservative treatment for stage 1 and 2 tibialis posterior dysfunction is largely based on the clinician's anecdotal evidence as the majority of the literature focuses on diagnosis, classification system, and operative options.
Alvarez et al. conducted a study involving non-operative treatment of stage 1 and 2 tibialis posterior tendon dysfunction which included prescription of orthotics and a rehabilitation program. (15) The rehabilitation program focused on specific strengthening exercises for the tibialis posterior, peroneals, tibialis anterior, and gastrocsoleus. Progression was aimed at achieving high repetitions of double and single leg heel raises in order to train the muscles for long-term endurance. This study found that 89% of patients responded to a regimen of orthotic use and supervised physical therapy. (15)
The patient in our case study was treated with medical acupuncture (4 points surrounding the injury) with electrical stimulation (IC-1107+ at 2 Hz frequency), thera peutic ultrasound with Traumeel (5 minutes, 50% duty cycle, 1 MHz frequency, 0.7 W intensity), ART[R] of gastrocnemius, soleus, and tibialis posterior muscles above and below the injury, and Graston Technique[c] with GT 6 and GT 2 posterior to the medial malleolus followed by ten minutes of ice and elevation. The intent of these treatments was to restore the proper blood supply to the muscle, reduce fibrotic tissue/adhesions and restore function to the muscle.
Graston Technique[R], also referred to as an augmented soft tissue mobilization technique, employs specially designed stainless steel instruments with bevelled edges to augment a clinician's ability to perform soft tissue mobilization. The instruments are utilized in a multidirectional stroking fashion applied to the skin at a 30[degrees]-60[degrees] angle at the treatment site. This application allows the clinician to detect irregularities in the soft tissue texture through the undulation of the gliding tools. (16,17) In addition to removing scar tissue adhesions, Graston Technique[R] is proposed to enhance the proliferation of extracellular matrix fibroblasts, improve ion transport and decrease cell matrix adhesions. (16,17)
Active Release Technique[R] therapy is utilized with the underlying understanding that the anatomy of the limbs has traversing tissues situated at oblique angles to one another that are prone to reactive changes producing adhesions, fibrosis and local edema and resultant pain and tenderness. (18,19) During Active Release Technique[R] therapy, the clinician applies a combination of deep digital tension at the area of tenderness and the patient actively moves the tissue through the adhesion site from a shortened to a lengthened position. (18,19)
Activity modifications were also prescribed, including a break from his running training while continuing cycling and swimming with the use of a pullbuoy. Tibialis posterior strengthening exercises of heel-ups with a tennis ball between the medial malleoli were prescribed. (14)
A gait analysis and a gait scan was also performed from which orthotics were prescribed and fitted before running was resumed. The prescription of orthoses for early stage tibialis posterior dysfunction is well supported in the literature. (5,6,9,14,15)
A wide variety of operative treatments have been reported for tibialis posterior tendon dysfunction. Stage 1 and 2 dysfunction is rarely treated operatively unless conservative management has failed, at which time debri dement and immobilization is considered. There are a variety of isolated soft-tissue procedures designed to compensate for a dysfunctional tibialis posterior tendon and these reconstructive surgeries or osteotomies are employed to improve alignment of the foot in stage 3 and 4 dysfunction. Recovery from reconstructive surgery is prolonged and an eventual return to asymptomatic unrestricted activities is unpredictable. (5)
Injuries to the lower leg/ankle are common in triathletes due to overuse mechanisms. These injuries are commonly reported during running and rarely reported during swimming. The patient in this case report presented with an acute injury to the lower leg/ankle as a result of a swimming injury. This patient was treated seven times consisting of medical acupuncture with electrical stimulation, Active Release Technique[R], Graston Technique[c], strengthening exercises, and gait analysis. He responded favourably to these conservative measures and was able to return to pre-injury status and resume triathlon training. Practitioners treating this type of injury could consider including the soft tissue techniques and modalities employed in our case for patients with lower leg strains and/or tibialis posterior injuries.
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(7) Prado MP, de Carvalho AE, Rodrigues CJ, Fernandes TD, Mendes AAM, Salomao O. Vascular density of the posterior tibial tendon: a cadaver study. Foot and Ankle International. 2006; 27(8):628-631.
(8) Uchiyama E, Kitaoka HB, Fujii T, Luo ZP, Momose T, Berglund LJ, An KN. Gliding resistance of the posterior tibial tendon. Foot and Ankle International. 2006; 27(9):723-727.
(9) Kohls-Gatzoulis J, Angel JC, Singh D, Haddad F, Livingstone J, Berry G. Tibialis posterior dysfunction: a common and treatable cause of adult acquired flatfoot. British Medical Journal. 2004; 329:1328-1333.
(10) Yeap JS, Singh D, Birch R. Tibialis posterior tendon dysfunction: a primary or secondary problem? Foot and Ankle International. 2001; 22(1):51-55.
(11) Johnson KA. Tibialis posterior tendon dysfunction. Clin Orthop. 1989; 239:196-206.
(12) Myerson MS. Adult acquired flatfoot deformity: treatment of dysfunction of the posterior tibial tendon. J Bone Joint Surg. 1996; 78A:780-792.
(13) Ness ME, Long J, Marks R, Harris G. Foot and ankle kinematics in patients with posterior tibial tendon dysfunction. Gait and Posture. 2008; 27:331-339.
(14) Tome J, Nawoczenski DA, Flemister A, Houck J. Comparison of foot kinematics between subjects with posterior tibialis tendon dysfunction and healthy controls. J Orthop Sports Phys Ther. 2006; 36(9):635-644.
(15) Alvarez RG, Marini A, Schmitt C, Saltzman CL. Stage I and II posterior tibial tendon dysfunction treated by a structured nonoperative management protocol: an orthosis and exercise program. Foot and Ankle International. 2006; 27(1):2-8.
(16) Sevier TL et al. Treating Lateral Epicondylitis. Sports Medicine. 1999; 28(5):375-380.
(17) Gehlsen GM et al. Fibroblast responses to variation in soft tissue mobilization pressure. Med Sci Sports Exerc. 1999; 31(4):521-5.
(18) Mooney V. Overuse Syndromes of the upper extremity: rational & effective treatment. J Musc Med. 1998; 15(8):11-18.
(19) Schiottz-Christensen B et al. The role of active release manual therapy for upper extremity overuse sindromes--a preliminary report. J Occup Rehab. 1999; 9(3):201-11.
Scott Howitt, DC, FCCSS(C), FCCRS(C) * Sarah Jung, DC Nicole Hammonds, DC
* Assistant Professor, Clinical Education, Canadian Memorial Chiropractic College, 6100 Leslie St., Toronto, Ontario M2H 3J1. Phone: (416) 482-2340 ext. 395 Fax: (416) 488-0470 Email: email@example.com
Table 1 Staging Classification Stage 1: defined as the absence of a fixed deformity of the foot or ankle. Patient typically presents with pain along the course of the tibialis posterior tendon and local inflammatory changes; however the tendon is of normal length and function. Stage 2: characterized by dynamic deformity of the hindfoot. The standing patient displays an increased degree of hindfoot valgus, apparent weakness of tibialis posterior function, "too many toes" sign, and inability to do a single leg heel rise; however patients still have a relatively normal arc of subtalar motion. Stage 3: patients have a fixed deformity of the hindfoot and it is not possible to reduce the talonavicular joint. Typically these patients also have an accompanying fixed forefoot supination deformity that is a compensatory change to accommodate the hindfoot valgus. Stage 4: consists of a stage 3 deformity with evidence of associated tibiotalar asymmetry because of the prolonged hindfoot valgus deformity. They may present with ankle arthritis.
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